Aneuploidy in the budding yeast Saccharomyces cerevisiae

Hello everyone,

My name is Zach Oppler and I’m a rising junior here at William and Mary. I’m a CAMS (biology track) and Psychology double major and I’m from Potomac, Maryland. This semester I worked in Dr. Murphy’s evolutionary genetics lab and that’s where I’ll be working this summer as well.

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New Developments Concerning the Circadian Rhythm Experiments




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The Application of Microwave Technology to Glaser-Hay Couplings

Hi everyone! My name is Sanjana Verma and I’m a rising senior (wow, it feels weird to type that) at the College. I’m from Northern VA (…who isn’t?), more specifically around the Chantilly/Centreville area, which is about 30-45min away from D.C. I’m a Biological Sciences major and a Religious Studies minor, and hope to be a doctor one day. I’m a student in Dr. Young’s lab and am really excited for this summer and what it may bring! Now a little bit about what I’ve done/plan to do…

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Woody Internship in CW!!! (Intro post)

Hello all,

My name is Emma Merrill, and I am extremely excited to be a recipient of the Woody Internship in Museum Studies for this summer! I will be working for the Colonial Williamsburg Foundation here in Williamsburg, hopefully learning more about 18th century art history, museum curation/exhibition, and doing a little research in the process.

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A Computational Model including Role of NF-κB in Multiple Sclerosis

Multiple Sclerosis (MS) is an autoimmune, neurodegenerative disease that cannot be cured. The disease causes the body’s own immune system to attack the axons of neurons, disrupting communication between the brain and the body. Those afflicted with Multiple Sclerosis slowly lose the ability to send signals down their axons. Consequently, they are no longer able to function or use those parts of their nervous system, and in many cases become immobile. The mechanism for axonal degradation is not properly understood, but a variety of pathways and factors have been discussed as contributing towards the degradation. NF-κB is a well-known transcription factor that can be cited as the “master switch” for immune response, and can be known to alter the behavior in immune cells’ response to injury and function. In the context of MS, NF-κB controls the macrophage phenotype that is expressed.

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